Includes bibliographical references and index.
|Statement||edited by James L.M. Ferrara, H. Joachim Deeg, Steven J. Burakoff.|
|Contributions||Ferrara, James L. M., 1952-, Deeg, H. Joachim 1945-, Burakoff, Steven J., 1942-|
|LC Classifications||RD123.5 .G73 1997|
|The Physical Object|
|Pagination||xviii, 824 p. :|
|Number of Pages||824|
|LC Control Number||96031581|
In most of the immunodeficient patients, a GVHD is fatal. Skin is the most commonly affected organ with a typical distribution of rash affecting soles and palms initially, progressing to head, face and body. However, GVHD is a rare disease because of an improvement in tissue matching by: Overview; GVHD occurs when significant numbers of functional donor T-cells are directly engrafted into a recipient or if donor T-cells can develop from engrafted Hematopoietic Stem Cells. GVHD is much more common in immunocompromised recipients and this is frequently the case in the context of bone marrow or solid organ transplantation in which the immune system . GVHD occurs when the donor’s T cells (the graft) view the patient’s healthy cells (the host) as foreign, and attack and damage them. Graft-versus-host disease can be mild, moderate or severe. In some cases, it can be life-threatening. Unless the patient’s donor is an identical twin, a patient receiving an allogeneic stem cell transplant. Acute graft-vs-host disease: pathobiology and management. Exp Hematol. ; – [Google Scholar] Nevo S, Enger C, Swan V, et al. Acute bleeding after allogeneic bone marrow transplantation: association with graft versus host disease and effect on survival. Transplant. ; – Cited by:
Graft vs. Host Disease 3rd Edition by James Ferrara (Editor), H. Joachim Deeg (Editor), Kenneth R. Cooke (Editor) & out of 5 stars 2 ratings. ISBN ISBN Why is ISBN important? ISBN. This bar-code number lets you verify that you're getting exactly the right version or edition of a book. 5/5(2). Acute graft-vs-host disease (GVHD) is a major obstacle to safe allogeneic hematopoietic stem cell transplantation (HSCT), leading to a significant morbidity and mortality. With detailed contributions from more than 40 leading authorities on the topic, this Third Edition comprehensively explores the immunobiology, pathophysiology, and clinical manifestations of graft-versus-host disease (GVHD)-offering sections revealing the most up-to-date research on immune activation and dysregulation, the pathophysiology of targetCited by: Pathology of Graft vs. Host Disease will serve as a very useful resource for physicians and researchers dealing with, and interested in, this challenging disease. It provides a concise yet comprehensive summary of the current status of the field that will help guide patient management and stimulate investigative : Hardcover.
Chronic graft-versus-host disease (GVHD) can affect eating, drinking and tooth brushing: So, with the mucosal disease, again, that lichenoid pattern of inflammation is really classic. The cheeks and the tongue are most common, lips are really frequently affected. Graft versus host disease (GVHD) is a complication that can develop after a person has had a bone marrow or stem cell transplant. In the disease, . Up to 70 percent of transplant recipients, such as Greg Grappone, develop acute graft-vs.-host disease, which crops up within the first few months of treatment, and 40 percent get chronic GVHD, the form that appears more than days post-transplant. As the name implies, the chronic condition can last for years — or a lifetime — and its symptoms range from mildly . With detailed contributions from more than 40 leading authorities on the topic, this Third Edition comprehensively explores the immunobiology, pathophysiology, and clinical manifestations of graft-versus-host disease (GVHD)-offering sections revealing the most up-to-date research on immune Price: $